Heat Exhaustion in Dogs

• Deep breathing or hyperventilation
• Anxiousness
• Deliriousness
• Dark red gums
• Restlessness
• Dizziness or confusion
• Thick saliva
• Inattentiveness
• Increased salvation followed by dry gums
• Weakness
• Very high temperature
• Diarrhea
• Vomiting

• If your pet dog is showing the signs of heat exhaustion, shift it too a cooler location, either in an air conditioned room or in a shaded area away from direct exposure of the sun.
• Once you have done this, pour cool water or apply ice pack on the dog to bring down his body temperature. Now, here you need to be careful, as applying ice pack on its back can damage its internal organs beyond repair. Hence, carry this procedure on the dogs belly.
• While suffering from heat exhaustion, there are chances that your dog may vomit the food that he has eaten. Under such circumstances, do not give him water to drink as he may vomit this too. Therefore, try cooling it by other methods and let it drink water only after the body temperature reaches 103 degrees.
• Once the temperature is brought back to normal and the signs of heat exhaustion in dog diminish, take your canine to the vet who will do a complete check up to see whether any long term effects have occurred.

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When to Use Ice When to Use Heat

• Infrared sauna: 160-180 degrees Fahrenheit, for 15-30 minutes
• Regular (Finnish wet or dry) sauna: 180-190 degrees Fahrenheit, for 10-20 minutes

• "Drastic" decrease in uric acid levels: High levels of uric acid are normally associated with gout, but it has been long known that people with high blood pressure, kidney disease, and people who are overweight often have elevated uric acid levels. When your uric acid level exceeds about 5.5 mg per deciliter, you have an increased risk for a host of diseases including heart disease, fatty liver, obesity, diabetes, hypertension, kidney disease, and more.

• Increase in glutathione: Glutathione is your bodys most powerful antioxidant, which keeps all other antioxidants performing at peak levels.

• Place an ice pack on your upper back and upper chest for 30 minutes per day (you can do this while relaxing in front of the TV for example)
• Drink about 500 ml of ice water each morning
• Take cold showers
• Immerse yourself in ice water up to your waist for 10 minutes, three times per week. (Simply fill your tub with cold water and ice cubes)

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Symptoms of a Dog in Heat

• The changes in the dogs may vary from quite mild to more severe. Some dogs may become more affectionate and clingy to their owner while others may seem a bit grumpy or fussy.
• Its quite unusual for your pet dog to undergo appetite changes, as very rarely the dog may go off her food or become hungry to the point of raiding the trash bin for discarded leftovers. But whatever may be the change, keep in mind as it can be significant sign that the heat period has begun.
• Another sign is the swelling of the vulva which can be noticed trailing up to the pelvic opening just below the anus. Along with the swelling, there will be bloody discharges from the vulva which is less during the initial few days but becomes a bit heavier till the mid-week.
• The dog will try to guard the vulva, either by tucking the tail between the leg or sitting down whenever another the male dog approaches the immediate area.

• Pinkish-tan lightened discharge from the vulva is the first sign for the onset of estrus.
• The initial swelling of the vulva subsides and it becomes soft enough for penetration.
• Since the female dog is now ready for fertilization, she will start flagging her tail and behave flirtatiously to invite male dogs.

• During this time, most of the swelling disappears but the vulva still remains slightly enlarged.
• Whether bred or not, the female dog now lacks the conditions to mate and is no longer interested in mating.
• The pinkish-tan vaginal discharge will turn red again but will gradually tapers off over the course of the final week.

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Heat Wave–Associated Vibriosis Sweden and Finland 2014 Volume 22 Number 7—July 2016 Emerging Infectious Disease journal CDC

Volume 22, Number 7—July 2016

Research

Heat Wave–Associated Vibriosis, Sweden and Finland, 2014

Craig Baker-Austin , Joaquin A. Trinanes, Saara Salmenlinna, Margareta Löfdahl, Anja Siitonen, Nick G.H. Taylor, and Jaime Martinez-Urtaza

Results

Figure 1. Monthly reportedVibrio infections in Sweden and Finland, May–December 2014. Beginning in July and increasing in August, reported infections spiked, corresponding with the heat wave in Scandinavia during that time.

A total of 89 Vibrio infections were reported in Sweden and Finland during the summer and autumn 2014, the largest yearly total number of cases, to our knowledge, identified in these countries. Infections were apparent across most Baltic coastal counties of Sweden and Finland. Numerous cases were reported at extreme subarctic regions, and as far north as >65°N, <100 miles (160 km) from the Arctic Circle. Reported infections began in July 2014 and peaked in August, before decreasing significantly in September (Figure 1). Infections were spread across persons of widely varying ages (range 3–93 years; median 36.2 years). In general, those infected were more commonly male (61 [67%] cases). One known fatality was noted: a V. cholerae non–O1/O139 infection reported in August 2014 from southern Sweden. Data on possible transmission was largely absent from the dataset from Finland; however, most cases in Sweden during 2014 occurred among persons who reported recreational exposure to seawater (e.g., the Baltic Sea) or lake water before infection (33 [78%] cases). Most (70 [77%]) infections were attributed to V. cholerae non–O1/O139; in 1 case, a ctx-negative O1 strain was reported. Other species reported were V. alginolyticus (3 cases), V. parahaemolyticus (4 cases), V. vulnificus (2 cases), V. mimicus (1 case), and unspecified Vibrio species (8 cases) (Table). Thirty-three (37%) infections were associated with ear or ear secretion isolations; however, for 17 (19%) of the 89 reported cases, Vibrio organisms were isolated directly from blood, suggesting more serious systemic disease progression.

Figure 2. Location of reportedVibrio infections in coastal areas, Sweden and Finland, 2014. The number of infections coupled with the extreme SST anomaly, particularly in northern latitude areas, is particularly noteworthy. SST,...

Figure 3. SST anomaly data for coastal areas of Sweden and Finland. A) Maximum SST anomalies during July and August 2014. The anomalies were substantially high throughout the region but especially in the...

The temporal and spatial distribution of reported cases corresponded closely with a highly anomalous heat wave in northern Finland and Sweden during July and August 2014, where SSTs in the northern Baltic exceeded all known long-term climatic and oceanographic records. A persistent and long-lasting period of high pressure occurred in northern Finland and Sweden beginning in May 2014, and this weather pattern persisted until mid-August. Concomitantly, SST in the Baltic Sea area was highly anomalous during July and August 2014; temperatures peaked toward the end of July. In some coastal regions, SSTs were ?10°C higher than the long-term average, indicating the extreme severity of this anomaly (Figure 2). Across the northern Baltic Sea area, SSTs were several degrees Celsius warmer than had been reported since the early 1980s. SSTs across large swathes of the Baltic and the Gulf of Bothnia area, in particular, had SSTs >18°C for several weeks beginning in mid-July and ceasing in mid-August (Figure 3). SST’s reported in the Gulf of Bothnia at the end of July were the most extreme reported during 1981–2016, exceeding 21.7°C on July 29, 2014, and with several days of temperatures >20°C. The observed SST anomaly during this period was also the largest ever seen in this dataset, encompassing almost 13,000 data points, with an anomaly of 9.79°C on July 29, 2014.

A statistical analysis between maximum SST and annual Vibrio cases using a generalized linear model showed that maximum SST explained a significant amount of the variability in cases (as determined by a significant reduction in the residual deviance from 120.55 to 42.16). The model predicted that, as the maximum SST increases, the number of annual number of cases also will increase significantly (? = 0.33002, SE = 0.08045,t = 4.102, p = 0.00343).

Discussion

Figure 4. Total reported Vibrioinfections in Finland and Sweden, 2005–2014. Foreign-acquired infections (where known) were omitted from the analyses. Epidemiologic data were gathered from public health agencies in Sweden and Finland (see...

Domestically acquired Vibrio infections are rare in northern Europe, and the spike in recorded cases of vibriosis reported in this region is particularly noteworthy. The cases in 2014 are the largest yearly total of reportedVibrio infections in Sweden and Finland, more than double the number of reported cases than in other recent years (Figure 4). In Sweden, 2014 was the warmest year on record since recordkeeping began in 1860; in Finland, 2014 was the second-warmest year on record (10,11). Across Finland, 50 days of hot summer weather (temperatures >25°C) were recorded during May–August, which is 14 days more than the long-term average (10). The large number of reported infections corresponded closely with an intense and northerly SST anomaly, suggesting that these unusual oceanographic and climatic conditions drove this episode of waterborne disease. A subsequent quantitative and statistical analysis of SST data from this region revealed 3 further observations: 1) the peak SSTs in late July 2014 were the most intense observed in the Bay of Bothnia; 2) the anomaly is the most intense in almost 35 years of climate data (1981–2015); and 3) the likelihood of such an event occurring based on recent climate data (1981–2015) is highly unlikely—the 2014 maximum observed temperature was significantly higher than the maximum expected based on the data for other years, and based on the distribution of maximum temperatures observed, a temperature this much higher than the mean would be expected only in 0.78% of years (once every 128 years).

Vibrio species such as V. cholerae grow preferentially in low-salinity warm water, and recreational exposure to water, which appears to have been responsible for a sizeable proportion of these reported infections, also increases substantially during heat waves. That 2014 followed several other recent heat wave years (e.g., 1994, 1997, 2003, 2006, and 2010), during which recorded domestically acquired Vibrio cases spiked in northern Europe (4,5), is particularly noteworthy. Previous epidemiologic analysis regarding the emergence of Vibrioinfections in the region (5) indicated that sustained SSTs >18°C were a notable risk factor, significantly increasing reported cases. The relation between maximum SST and annual Vibrio case occurrence analyzed by using generalized linear model–based methods demonstrated similarly to previous studies in the region (4) that maximum temperature correlates highly with risk, and cooler years (e.g., 2005, 2007, and 2012) indicate lower levels of reported infections than heat wave years (e.g., 2006, 2010, and 2014). In our study, the observation that a sizeable proportion of described cases were reported in subarctic latitudes (>65°N) and within 100 miles (160 km) of the Arctic Circle is striking. Ten V. cholerae infections were reported above 63°N, of which 6 cases were identified in the Oulo area (?65°N). The cases recorded here are, to our knowledge, the most northerly reported instances of vibriosis documented, exceeding previous studies where cases have been reported at high latitudes, such as Alaska (9) and previously in northern Europe (5).

Disease data, such as those reported here, often are sporadic and usually grossly underreported. Likewise, a major limitation of our investigation was the lack of detailed trace-back epidemiologic data, which limits the assessment of exposure and subsequent risk. For many reported cases, data about prior exposure (e.g., specific information about the timing and location of recreational exposure to water) and subsequent routes of transmission were absent. However, almost without exception, cases from Finland and Sweden were reported in coastal rather than inland medical centers. Second, when prior transmission information was available from confirmed cases, most patients reported exposure to seawater in the days before symptom onset. These 2 factors, coupled with the striking climatic and oceanographic conditions during summer 2014, suggest that exposure to seawater was largely responsible for these episodes of disease emergence. The limitations underscore the need for a centralized system of surveillance and reporting. In the United States, the Centers for Disease Control and Prevention’s COVIS (Cholera and Other Vibrio Illness Surveillance) maintains a national database of vibriosis that contained detailed epidemiologic and transmission route information (12). A similar centralized reporting, monitoring, and surveillance system would greatly enhance risk assessment and risk management of vibriosis in Europe. Across the region, and with the exception of toxigenic V. cholerae infection, vibriosis is not a notifiable disease (5). Given that these rare waterborne infections appear to have emerged and increased in northern Europe recently (13) (e.g., 1994, 2006, 2014), this event underlies the need for clinicians to identify possible exposure to seawater. This event is particularly relevant for patients who have a history of conditions where progression of vibriosis to systemic infection is more likely, including diabetes, immune disorders, and liver dysfunction.

Climatic anomalies, such as the heat wave conditions during summer 2014 in northern Europe, appear to be responsible for restructuring the geographic distribution of waterborne infectious diseases and resulted in major and far reaching consequences for the identification, treatment, and management of these pathogens. The greater number and intensity of large heat wave events in northern Europe during the past 20 years or so (1994, 1997, 2003, 2006, 2010, 2014) further highlights the need for improved epidemiology and reporting, coupled with enhanced diagnostic capability in clinical settings to manage and ameliorate risk.

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